Diagnosis

Eosinophilic esophagitis

A chronic immune/antigen-mediated inflammatory disorder causing eosinophilic infiltration of the esophagus and esophageal dysfunction.

Etiology

Cause [zrk]

Patophysiology [zrk]

  1. There are no physiologic eosinophils in the esophagus

  2. Eosinophils present in the esophagus in response to various stimuli or antigen

  3. Mucosal inflammation --> remodeling of esophageal mucosa --> fibrosis

  4. Smooth muscle dysfunction --> dysphagia, epigastric/chest pain, dyspepsia, and food impaction

  5. Disease course:

    • Spontaneous remission with or without treatment

    • Waxing and waning course

    • Progressive course leading to strictures

Risk factors [wfm]

Epidemiology

Prevalence per 100.000 [wfm][dwy][im6][siq][psb]

Epidemiology chart for Prevalence

Incidence per 100.000 [wfm][dwy][im6][siq][psb]

Epidemiology chart for Incidence

Symptoms & findings

Symptoms

Diarrhea, Dyspepsia, Dysphagia, Failure to thrive, Feeding difficulty, Heartburn, Nausea, Vomiting

Clinical findings

Esophageal stricture, Esophageal ulcer, Gastroesophageal Reflux Disease

Anamneses

None listed.

Localized findings

Pain
Radiates
Regio epigastricaRetrosternal
Onset
Subacute (hours)Gradual (days)
Pattern
ConstantRecurring
Provoked by
EatingPostprandial
Quality
Burning
Severity
Mild (1-3)Moderate (4-7)

Approach

Diagnostic criteria [im6]

Treatment

  1. Dietary therapy: exclusions of antigenic foods [zrk]

    • If unknown allergenic food or agent: a trial of food elimination diet

    • Common allergenic food: milk, wheat, peanut, egg, soy, and seafood/shellfish

  2. Proton pump inhibitors 20-40 mg daily or twice daily

  3. Topical steroids: Budesonide 1-2 mg per day

  4. Systemic steroids: Prednisone 2 mg/kg/day (max 60mg/day)

  5. Endoscopic dilation in cases with strictures

  6. Immunsuppresion:

    • Monoclonal antibody: Dupilumab [ryh]

Differential diagnoses

Achalasia, Celiac disease, Crohn disease, Drug side effects, Eosinophilia, Eosinophilic gastroenteritis, Gastroesophageal reflux disease, Graft-versus-host disease, Mixed connective tissue disease, Pemphigus vulgaris, Vasculitis


References

[1] Roussel JM, Pandit S. Eosinophilic Esophagitis. [Updated 2023 Aug 8]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2025 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK459297/

[2] Dellon ES. Epidemiology of eosinophilic esophagitis. Gastroenterol Clin North Am. 2014 Jun;43(2):201-18.

[3] Arias Á, Pérez-Martínez I, Tenías JM, Lucendo AJ. Systematic review with meta-analysis: the incidence and prevalence of eosinophilic oesophagitis in children and adults in population-based studies. Aliment Pharmacol Ther. 2016 Jan;43(1):3-15.

[4] Hahn JW, Lee K, Shin JI, Cho SH, Turner S, Shin JU, Yeniova AÖ, Koyanagi A, Jacob L, Smith L, Fond G, Boyer L, Lee SW, Kwon R, Kim S, Shin YH, Rhee SY, Moon JS, Ko JS, Yon DK, Papadopoulos NG. Global Incidence and Prevalence of Eosinophilic Esophagitis, 1976-2022: A Systematic Review and Meta-analysis. Clin Gastroenterol Hepatol. 2023 Dec;21(13):3270-3284.e77.

[5] Soon IS, Butzner JD, Kaplan GG, deBruyn JC. Incidence and prevalence of eosinophilic esophagitis in children. J Pediatr Gastroenterol Nutr. 2013 Jul;57(1):72-80.

[6] Dellon ES, Jensen ET, Martin CF, Shaheen NJ, Kappelman MD. Prevalence of eosinophilic esophagitis in the United States. Clin Gastroenterol Hepatol. 2014 Apr;12(4):589-96.e1.

[7] https://emedicine.medscape.com/article/1610470

[8] Furuta G.T., Katzka D.A.: Eosinophilic esophagitis. NEJM 2015; 373: pp. 1640-1648.

[9] Buckmeier, Bridget K. et al. The Incidence and Prevalence of Eosinophilic Esophagitis. The Journal of Allergy and Clinical Immunology 121 (2008).

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