Diagnosis

Toxic nodular goiter

Excessive production of thyroid hormones from functionally autonomous thyroid nodule which does not require TSH:

  • Nodular goiter: ≥ 1 nodule

  • Multinodular goiter: ≥ 2 nodules

  • Diffuse goiter: a smooth and uniformly enlarged thyroid gland

Also known as: Plummer Disease, Plummer's disease, Toxic adenoma, Toxic multinodular goitre

Etiology

Cause [cpt]

Pathophysiology [cpt][9kz]

  1. Chronic low-grade stimuli:

    • Iodine deficiency --> decreased T4 --> compensatory diffuse thyroid hyperplasia

    • Long-lasting elevated TSH --> diffuse thyroid hyperplasia

  2. Increased risk of mutation in the TSH receptor -> continuously active TSH receptor

  3. Toxic adenoma: autonomous nodule(s) independent of pituitary TSH feedback)

  4. Clinical presentation:

    • Heat intolerance 

    • Weight loss 

    • Tremors 

    • Palpitations, tachycardia, atrial fibrillation

    • Increased bowel frequency 

    • Menstrual irregularity 

    • Sweaty palms, excessive sweating

Complications [cpt]

Epidemiology

Incidence per 100.000 [cpt][8dw][qgp][1wr][rso][ybe]

Epidemiology chart for Incidence

Symptoms & findings

Symptoms

Diarrhea, Fatigue, Heat intolerance, Hyperhidrosis, Hyperkinesis, Increased appetite, Irritability, Muscle atrophy, Muscle weakness, Palpitations, Restlessness, Tachycardia, Tremor, Weight loss, Wide pulse pressure

Clinical findings

Atrial fibrillation, Decreased TSH, Elevated T3, Elevated T4, Gynecomastia, Increased iodide uptake

Anamneses

None listed.

Localized findings

Swelling
Radiates
Collum
Onset
Gradual (days)
Pattern
ConstantIncreasing

Approach

Treatment

  1. Subclinical hyperthyroidism --> monitored without intervention [cpt]

  2. Surgery: lobectomy or total thyroidectomy is the mainstay therapy [ybe]

    • Complications: damage to the recurrent laryngeal nerves, hypoparathyroidism

    • Recurrence rate: near zero

  3. Radioactive iodine

    • Recurrence rate: 15–25%

  4. Antithyroid drugs: if neither surgery or radioactive iodine is possible

Differential diagnoses

Graves disease, Hashimotos thyroiditis, Osteoporosis, Riedels fibrosing thyroiditis, Struma ovarii, Subacute thyroiditis, Thyroid cancer, Toxic thyroid adenoma


References

[1] Khalid N, Can AS. Plummer Disease. Updated 2023 Mar 11: https://www.ncbi.nlm.nih.gov/books/NBK565856/

[2] Knobel M. Etiopathology, clinical features, and treatment of diffuse and multinodular nontoxic goiters. J Endocrinol Invest. 2016 Apr;39(4):357-73. doi: 10.1007/s40618-015-0391-7. Epub 2015 Sep 21.

[3] Censi S, Salmaso L, Ceccato F, Manso J, Fedeli U, Saia M, Mian C. Hyperthyroidism incidence in a large population-based study in northeastern Italy. Endocr Connect. 2023 Nov 14;12(12):e230292.

[4] Baltisberger BL, Minder CE, Bürgi H. Decrease of incidence of toxic nodular goitre in a region of Switzerland after full correction of mild iodine deficiency. Eur J Endocrinol. 1995 May;132(5):546-9.

[5] Abraham-Nordling M, Bystrom K, Torring O, Lantz M, Berg G, Calissendorff J, et al. Incidence of hyperthyroidism in Sweden. European journal of endocrinology / European Federation of Endocrine Societies. 2011;165(6):899–905.

[6] Abraham-Nordling M, Törring O, Lantz M, Hallengren B, Ohrling H, Lundell G, Calissendorff J, Jörneskog G, Wallin G. Incidence of hyperthyroidism in Stockholm, Sweden, 2003-2005. Eur J Endocrinol. 2008 Jun;158(6):823-7.

[7] Sjölin, G., Watt, T., Byström, K. et al. Long term outcome after toxic nodular goitre. Thyroid Res 15, 20 (2022).

[8] http://emedicine.medscape.com/article/120497 (2014-01-02); [Medscape]

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